MTRR A66G Mutation Basics
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The MTR A2756G mutation is a defect of the methionine synthase gene. A mutation of the MTR A2756G gene results in up-regulation of the enzymatic pathway that converts homocysteine into methionine. This enzyme utilizes the methylated form of vitamin B12 to function, and the up-regulation of this enzyme can deplete methyl-B12 levels rapidly. This often results in a deficiency of methyl-B12, which can lead to elevated homocysteine levels.
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MTRR A66G Mutation Basics. MTRR A66G Mutation Basics. The MTRR A66G mutation is a defect of the methionine synthase reductase gene. A mutation of the MTRR A66G gene results in poor methylation of vitamin B12. Serum B12 levels may be normal in MTRR A66G or - individuals; however, the under-methylation of vitamin B12 may result in elevated homocysteine levels. If you have high homocysteine, it may be due to the MTRR A66G mutation. 199 Once you receive your results, go to. Because vitamin B12 and.CONTENT
This site had the following on the homepage, "The MTRR A66G mutation is a defect of the methionine synthase reductase gene." Our analyzers noticed that the web page said " A mutation of the MTRR A66G gene results in poor methylation of vitamin B12." The Website also stated " Serum B12 levels may be normal in MTRR A66G or - individuals; however, the under-methylation of vitamin B12 may result in elevated homocysteine levels. If you have high homocysteine, it may be due to the MTRR A66G mutation. 199 Once you receive your results, go to."VIEW MORE WEB SITES
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